Recently, it has been proposed a unified definition of both acquired and lifelong premature ejaculation (PE) as a male sexual dysfunction characterized by a reduction of latency time, inability to delay ejaculation, and negative personal consequences. [1] However, acquired premature ejaculation (APE) has peculiar characteristics (demographic differences: men with APE are usually older, presence of comorbidities and greater Intravaginal ejaculation latency time [IELT]) including the frequent association with the urogenital tract inflammations. [1] Previously, we reported that patients with male accessory gland infection (MAGI) have a high frequency of sexual dysfunction, detectable through the application of a dedicated questionnaire (SI-MAGI = structured interview about MAGI) and PE represents one of these dysfunctions. [2] Usually in the literature, it has been reported the association between APE and chronic P (prostatitis), [3] however, the P represents only one of the three diagnostic categories of MAGI: P (prostatitis), PV (prostato- vesiculitis), PVE (prostato-vesciculo-epididymitis). [4] A low number of evidences concerning the frequency of PE in patients with epididymitis [5] and chronic vesiculitis, [2] this aspect appears in contrast with some important physiological aspects, such as: the contractile function of the epididymis [6] and the role of the seminal vesicles in the production of the seminal plasma. [7] Chronic inflammation of the epididymis and seminal vesicles can also be evaluated through US (ultrasound) examination and in the past our group has published the US criteria for the diagnosis of MAGI. [8] On the basis of these premises, the aim of this study was to evaluate the presence of peculiar US features in patients with MAGI and APE associated (MAGI-APEpos) compared to patients with MAGI without PE (MAGI-PEneg).

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